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How is Hashimoto's thyroid treated?
2. In case of hypothyroidism, thyroid hormone replacement therapy is needed, and Eupatorium adenophorum is the first choice;
3. In case of hyperthyroidism, the principle of treatment is similar to that of inflammatory hyperthyroidism, and no treatment is needed. Propranolol can be given symptomatic treatment when the symptoms are obvious.
Hashimoto's thyroiditis is an autoimmune thyroiditis with a large number of lymphocytes, also known as chronic lymphocytic thyroiditis. This disease was discovered by Japanese Hashimoto Hakaru in 19 12, so it was named Hashimoto's thyroiditis (HT or AIT) for short. Hashimoto's goiter is often called Hashimoto's goiter because the thyroid gland of Hashimoto's thyroiditis is located in front of the neck. Hashimoto's disease is an autoimmune thyroid disease. Its pathological and immunological changes are similar to Grave's hyperthyroidism, that is, on the basis of genetic defects and genetic susceptibility, stress reactions such as mental factors, overwork, infection, environmental pollution and unreasonable diet structure (such as high iodine diet) can cause or aggravate autoimmune reactions and induce Hashimoto's thyroiditis. Therefore, Graves' hyperthyroidism and Hashimoto's thyroiditis can be transformed accordingly. Of course, more Graves' thyroiditis can be transformed into Hashimoto's thyroiditis.
The pathological mechanism of Hashimoto's thyroiditis is that autoimmune reaction produces autoantibodies. For example, anti-thyroid peroxidase antibody (TPOAb) and anti-thyroglobulin antibody (TGAb) form immune complexes with antigens. Cytotoxicity, immune mediation, immune complex, lymphocyte toxicity, etc. Dependent antibodies act on thyroid follicular epithelial cells, forming immune inflammation and inflammatory cell swelling, forming goiter in different degrees. A few patients with Hashimoto's thyroiditis have no goiter. Hashimoto's disease has a high incidence. It is reported in the literature that 5% of patients in a certain area have HT through repeated antibody tests. In our own long-term clinical work, Hashimoto's disease accounts for 38.9% of the total number of outpatients with thyroid diseases based on several antibody tests and statistics of thyroid cytology. Therefore, it is suggested that clinicians should check thyroid antibodies at the same time when checking T3 and T4 for thyroid patients, so as to avoid missed diagnosis. Patients with Hashimoto's thyroiditis should also ask a doctor to check thyroid antibodies.
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